Lecithin has been shown in the mouse to effectively block addiction to cocaine in mice.
"Soy phosphatidylcholine (SPC)"
"We propose that SPC can be useful as a selective inhibitor to suppress the TLR4-mediated inflammatory signaling"

Naloxone has been know to cause rapid detoxification of opioid addicts and has been used for decades as the primary treatment for opioid overdoses in ER's. It simply targets the opioid receptor sites and blocks them.

I do not understand how co-formulating a drug (morphine) with an antagonist that prevents the opioid from acting could actually cause an increase in pain relief. It 'should' cause the combination to be useless and have no effect.

40-year old news. Naloxone has been used for decades to counter the effects of opiate overdose. This was one of the first applications for the drug, developed in the 1960s.
It is a standard item in OD kits used by emergency responders. Naloxone is usually injected IV for fastest action. The drug acts within a minute, and may last up to 45 minutes. It blocks the cellular receptors used by opioids and endorphins, rendering the drugs totally ineffective.

Looks like Orexo in Sweden is working with such a drug since long time ago

Naloxone and (plus)-Naloxone are different. (See e.g. Wikipedia.)

Theoretically, blocking the TLR4/dopamine pathway might only affect the the psychological component of opioid dependence but have no effect on the physiological component, which may be due to unrelated functional mu-opioid receptor down-regulation mechanisms. This may limit any impact in general medical or palliative care, save for psychiatric substance abuse settings.